Anti-Mitofusin-1 Antibody (N111/24)

Our Anti-Mitofusin-1 mouse monoclonal primary antibody from NeuroMab is produced in-house from hybridoma clone N111/24. It is KO validated, detects mouse and rat Mitofusin-1, and is purified by Protein A chromatography. It is great for use in ICC, WB.



SKU: 75-162

Volume: 100 µL
1-2 business days
Price:
Sale price$319.00

Product Specific References for Applications and Species

Immunocytochemistry: Human
PMID Dilution Publication
323155971:100Herkenne, S., et al. 2020. Developmental and Tumor Angiogenesis Requires the Mitochondria-Shaping Protein Opa1. Cell Metabolism, 987-1003.
Western Blot: Human
PMID Dilution Publication
323155971:1000Herkenne, S., et al. 2020. Developmental and Tumor Angiogenesis Requires the Mitochondria-Shaping Protein Opa1. Cell Metabolism, 987-1003.
Western Blot: Mouse
PMID Dilution Publication
354141221:500O’Connor, T.N., et al. 2022. Endurance exercise attenuates juvenile irradiation-induced skeletal muscle functional decline and mitochondrial stress. . Skeletal Muscle, 45306.
30591411not listedMoore, T.M., et al. 2019. The impact of exercise on mitochondrial dynamics and the role of Drp1 in exercise performance and training adaptations in skeletal muscle. Molecular Metabolism, 51-67.
25497302not listedElezaby, A., et al. 2015. Mitochondrial remodeling in mice with cardiomyocyte-specific lipid overload.. Journal of Molecular and Cellular Cardiology, 275-283.
24949970not listedSoleimanpour, S.A., et al. 2014. The diabetes susceptibility gene Clec16a regulates mitophagy.. Cell, 1577-1590.
23695218not listedDolinsky, V.W., et al. 2013. Both aerobic exercise and resveratrol supplementation attenuate doxorubicin-induced cardiac injury in mice.. American Journal of Physiology. Endocrinology and Metabolism, E243-253.
230281131:1000Chao, L.C., et al. 2012. Skeletal muscle Nur77 expression enhances oxidative metabolism and substrate utilization.. The Journal of Lipid Research, 2610-2619.
225117811:1000Ngoh, G.A., et al. 2012. Loss of mitofusin 2 promotes endoplasmic reticulum stress.. The Journal of Biological Chemistry, 20321-30332.
220371951:1000Papanicolaou, K.N., et al. 2012. Cardiomyocyte deletion of mitofusin-1 leads to mitochondrial fragmentation and improves tolerance to ROS-induced mitochondrial dysfunction and cell death. American Journal of Physiology. Heart and Circulatory Physiology, H167-H179.

Bulk Order Anti-Mitofusin-1 Antibody

Related Products

Recently Viewed